MS Tissue-signaling research
Angiogenesis & VEGFR2
Angiogenesis and fibroblast migration are cellular processes measured in non-clinical models. Compounds in this family are cataloged for VEGFR2–nitric-oxide signaling and actin-driven migration markers. All work is in-vitro or pre-clinical and does not describe a human outcome.
MB Metabolic
Incretin signaling
Incretins are gut-derived hormones — chiefly GLP-1 and GIP — that potentiate glucose-dependent insulin secretion. The triple agonist in this family is studied in metabolic models for how combined GIP, GLP-1 and glucagon receptor engagement influences insulin signaling, energy expenditure and satiety circuits. Findings are pathway-level and pre-clinical.
GH GH Secretagogue
The growth-hormone axis
The GH axis is governed by two complementary receptors on pituitary somatotrophs — the GHRH receptor and the ghrelin / GHS receptor. The GHRH analogue in this family is studied for how it stimulates pulsatile GH release in endocrine research models. Research is confined to receptor-binding and release dynamics.
CM Cellular & Mitochondrial
Mitochondrial signaling
Mitochondria do more than produce energy; they also send signals that shape how a cell responds to metabolic stress. The mitochondrial-derived peptide in this family is studied for AMPK-pathway activation and for its stress-induced role in nuclear gene regulation in metabolic models. None of this is an anti-aging or health claim.
NP Neuropeptide
Neurotrophic & cognition
Neurotrophic factors such as BDNF and NGF support the maintenance and plasticity of neurons. The ACTH-derived heptapeptide in this family is studied in cognition and neuroprotection models for its modulation of those factors and of monoaminergic signaling. All work is in cell and animal models and makes no cognitive claim.
CS Skin & Cosmetic
ECM & collagen
The extracellular matrix (ECM) is the scaffold of collagen, elastin and glycosaminoglycans that surrounds cells. The copper-binding tripeptide in this family is studied for how it delivers copper and shifts fibroblast gene-expression tied to matrix remodeling. Findings come from cultured-cell and skin-research models only.